Introduction: Reframing Addiction through Biophysics
Addiction is often viewed as a behavioral or psychological issue, yet its origins are deeply biophysical. At its core, addiction is a consequence of mitochondrial dysfunction, redox imbalance, and circadian disarray. These cellular disruptions impair the brain's ability to produce and regulate key endogenous chemicals: dopamine, serotonin, beta-endorphin, oxytocin, DMT, cannabinoids, and steroid hormones. Without sufficient natural biochemical flow, the brain craves substitutes: artificial dopamine hits from technology, drugs, stimulants, alcohol, and other maladaptive coping mechanisms.
As Dr. Gabor Maté has emphasized, "The opposite of addiction is not sobriety. The opposite of addiction is connection." From a biophysics perspective, that connection must include both human resonance and electromagnetic coherence with our natural environment.
Part I: Addiction as a Biophysical Crisis
The addicted brain is not merely misfired; it is metabolically underpowered. Low mitochondrial redox, poor light signaling, disrupted calcium ion flow, and EMF-induced voltage stress all converge to impair the body’s internal reward circuitry.
What is the addicted brain deprived of?
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Full-spectrum sunlight → necessary for POMC cleavage into β-endorphin, ACTH, α-MSH
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Circadian-timed hormone and neurotransmitter synthesis → leptin, insulin, cortisol, serotonin, dopamine, melatonin
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Physical connection (oxytocin and endorphin release through touch and bonding)
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Safe electromagnetic environments (non-native EMFs dehydrate structured water, disrupt neurotransmitters)
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Quality consistent sleep, optimal breathing mechanics, and parasympathetic tone
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Empowered, inspiring and resonating connection
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Docosohexaenoic acid (DHA) from seafood
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Structured wáter
What is it overloaded with?
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Artificial blue light → excessive dopamine release, POMC dysregulation, melanopsin damage
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Anthropogenic nnEMFs → calcium efflux, low oxygen signaling, and oxidative stress
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Stimulants and digital media → hijack dopamine circuits and overstimulate PVN and limbic system
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Environmental toxins → Light, food, water, chemicals, etc.…
When these systems break down, the habenula nucleus the brain’s mood and addiction center suffers, cutting us off from joy, connection, and empathy. The result is chronic contraction, emotional looping, and dependence.

Part II: Addiction Pathways and the Role of Light
Sunlight, particularly UVA and IR-A light, catalyzes the body’s most powerful opioid: β-endorphin. This molecule, derived from the POMC gene in response to UV exposure, is released in small daily doses to generate wellbeing, pain reduction, and bonding.
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Morning UVA exposure triggers β-endorphin and dopamine, creating a mild natural addiction to sunlight itself (Fell et al., 2014)
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Infrared light (620–950 nm) increases mitochondrial ATP output with minimal ROS supporting the body’s energy levels and reducing feelings of addiction.
On the other hand:
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Blue light (without balancing IR) overstimulates dopamine and glutamate, leading to addiction-like cycles
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Artificial technology screens and LED/Fluorescent lighting lack the red/infrared needed to counteract ROS, depleting dopamine and BDNF
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Wireless radiofrequency and microwave radiation (Wi-Fi, Bluetooth, 4G, 5G, 6G, Virtual Reality Headsets, Wireless headphones and so on, raise blood glucose, dehydrate the body and weaken mitochondrial function, pushing the brain into short-term rewards and addictive behaviours.
The Opioid Crisis and Artificial Light
Exogenous opioids mimic the role of endorphins but fail to match their regulation and feedback signaling. Chronic use leads to vasopressin disruption, hyponatremia, gut paralysis, and brain fog. Blue light and technology exposure act as surrogate dopaminergic triggers, mimicking opioid effects and worsening dependency.
Neurotransmitter Release and Biophysical Disruption in Addiction
Calcium ions are vital for neurotransmitter release at synapses, acting as critical messengers in the brain's communication system. When this finely tuned system is disrupted by artificial light (particularly blue light) or non-native EMFs (nnEMFs) the calcium signaling pathways become dysregulated. This disturbs the second messenger systems and voltage-gated calcium channels, altering the timing and quantity of neurotransmitter release. Overactivation of NMDA receptors (glutamate receptors) due to this dysregulation can result in excitotoxicity, neuroinflammation, and mitochondrial stress contributing to the emotional instability, impulsivity, and dependency cycles seen in addiction. Furthermore, heavy metals can disrupt NMDA receptor activity and amplify these effects, intensifying the reward-seeking behaviors that characterize substance or behavioral addiction. These environmental disruptions alter the electrochemical harmony of the brain, priming it for addictive patterns and weakening its natural feedback systems like dopamine modulation and circadian hormonal cycling.
Part III: Gabor Maté, the Limbic System, and the Root of Addiction
Gabor Maté views addiction not as a choice but as a cry for regulation. From a biophysics standpoint, this means a cry for coherence. The limbic system and frontal lobes lose electromagnetic alignment when deprived of solar cues, meaningful connection, and nutrient flow. Emotional trauma rewires the pain body, shaping perception through survival filters, not presence.
Neurobiochemical insight:
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Trauma distorts electromagnetic imprinting in water (memory) within the cerebral spinal fluid (CSF). Make yourself more resistant to trauma by having more energy within your living system, then it won’t land so hard
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Circadian mistiming reduces BDNF and endorphins (mismatch of light during day and dark at night)
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Melanin and hemoglobin, our 2 key natural semiconductors, lose their conductivity without full-spectrum light
Part IV: Reversing Addiction through Natural Signals
Healing from addiction isn’t just about removing the harmful—it’s about restoring the environment that nourishes the whole system:
|
Natural Signal |
Biochemical Trigger |
Addiction Defense |
|
Sunrise light (UVA/IR) |
β-endorphin, dopamine |
Restores circadian pleasure |
|
Barefoot grounding |
Negative ions, voltage reset |
Stabilizes emotional charge |
|
Cold exposure |
Norepinephrine and dopamine boost, mitochondrial uncoupling |
Reduces inflammation & anxiety |
|
Human touch |
Oxytocin |
Connects and calms |
|
Exercise |
Endorphins, BDNF |
Neuroplasticity, resilience |
|
Music, Art, Flow, seafood & egg yolks |
Dopamine, serotonin |
Creative connection, purpose & happiness |
Addiction to Light: Nature's Design
There’s a reason nature made us addicted to sunlight. Beta-endorphin levels rise with sun exposure and decrease without it. This natural reward cycle promotes daily outdoor exposure, skin thickening, dopamine regulation, and serotonin-melatonin synthesis. Without this loop, humans become susceptible to synthetic heights and perpetual searching.
Just like we need light, we also need darkness and many struggling with addiction rarely experience real darkness at night. This is just as important to allow the brain and body regeneration time, where leptin, melatonin and growth hormone can power autophagy, the cellular renewal program, allowing a clear mind, energized body and a feeling of optimism in the early morning upon waking.
Conclusion: A Higher Structure for the Addicted Brain
Addiction is not a moral failure. It is the failure of the environment to meet the electromagnetic, biochemical, and emotional needs of the human being. Through restoring full-spectrum light exposure, mitochondrial redox, and social connection, the brain can rewire and re-energize.
The solution is not to fight addiction but to build a life more compelling than addiction itself. To replace the hijacked dopamine loops with habits and experiences that nourish the brain, recharge the mitochondria, and reconnect the soul to the body.
Let us shift from synthetic highs to cellular wholeness, where abundant stored electrical charge sits. Let us get addicted to sunrise, to resilience, to remembering who we are when our inner nature lives in coherence with nature.
References
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Fell, G. L., Robinson, K. C., Mao, J., Woolf, C. J., & Fisher, D. E. (2014). Skin β-Endorphin Mediates Addiction to UV Light. Cell, 157(7), 1527–1534. https://doi.org/10.1016/j.cell.2014.04.032
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Martin, L. F., Cheng, K., Washington, S. M., et al. (2023). Green Light Exposure Elicits Anti-Inflammation, Endogenous Opioid Release and Dampens Synaptic Potentiation to Relieve Post-Surgical Pain. The Journal of Pain, 24(3), 509–529. https://doi.org/10.1016/j.jpain.2022.10.011
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Tejeda, H. A., & Bonci, A. (2014). Shedding "UV" Light on Endogenous Opioid Dependence. Cell, 157(7), 1500–1501. https://doi.org/10.1016/j.cell.2014.06.009
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Zubieta, J. K., Smith, Y. R., Bueller, J. A., et al. (2001). Regional Mu Opioid Receptor Regulation of Sensory and Affective Dimensions of Pain. Science, 293(5528), 311–315. https://doi.org/10.1126/science.1060952
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Winters, B. L., Gregoriou, G. C., Kissiwaa, S. A., et al. (2017). Endogenous Opioids Regulate Moment-to-Moment Neuronal Communication and Excitability. Nature Communications, 8, 14611. https://doi.org/10.1038/ncomms14611
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Peciña, M., Karp, J. F., Mathew, S., et al. (2019). Endogenous Opioid System Dysregulation in Depression: Implications for New Therapeutic Approaches. Molecular Psychiatry, 24(4), 576–587. https://doi.org/10.1038/s41380-018-0117-2
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Martin, L. F., Moutal, A., Cheng, K., et al. (2021). Green Light Antinociceptive and Reversal of Thermal and Mechanical Hypersensitivity Effects Rely on Endogenous Opioid System Stimulation. The Journal of Pain, 22(12), 1646–1656. https://doi.org/10.1016/j.jpain.2021.05.006
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ScienceDirect. (2020). Blue Light and Dopamine Imbalance Linked to Parkinson’s Disease. https://doi.org/10.1016/j.neuroscience.2020.02.047
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MedicalXpress. (2019). Optimum Amount of Dopamine Supports Cognitive Ability. https://medicalxpress.com/news/2019-02-optimum-amount-dopamine-cognitive-ability.html
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Nature. (2005). Light Signaling and Hypothalamic Regulation of Sleep and Circadian Rhythm. https://www.nature.com/articles/nature04284
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JID. (2015). UVB Light and POMC System Activation in the Eye. https://www.jidonline.org/article/S0022-202X(15)30116-0/fulltext




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