A New Perspective on what medicine has missed in Parkinson’s Disease (PD) and neurodegeneration

Introduction and Background

The brain can be healed from an early neurodegenerative state using visible and non-visible light exposure on the eyes and skin. More recent research has showed the brains remarkable ability to regenerate if given the opportunity and we are experts in providing that opportunity for a neurodegenerative brain.

In 2022 a Parkinson’s Foundation-backed study became the most comprehensive assessment of Parkinson’s Diseases (PD) incidence in North America based on five epidemiological (the study of a disease and its impact) sets of data to count the number of diagnoses in 2012. Prior PD incidence rates, based on smaller studies, were estimated to be in the 40,000 – 60,000 people range per year. The new incidence rate is 1.5 times higher at nearly 90,000 cases annually. 40 years ago, the prevalence of PD was estimated to be around 1,500 people in North America diagnosed with PD each year. PD is dramatically on the rise and can almost be seen to have emerged out of nowhere 40 years ago affecting 1 in every 220,000 Americans each year, to now impacting 1 in every 3,500 Americans every year. This sharp increase seems alarming but makes total sense to us, and we’ll explain why.[9]

The way we live has dramatically changed in the past 40 years with much more indoor living, wide-spread technology use and a lack of emphasis on sleep. These and many more changes have contributed significantly to the changes we experience in our biology including the changes we see in the brain with respect to PD. Simply by changing the spectrum of visible and non-visible light we live under, we can change our brain, nervous system, endocrine system and immune system to name a few. We at Biospectral Systems are up to date and dare we say ahead of the science when it comes to explaining most modern neolithic diseases, including PD. We are very experienced with repairing brains with visible and non-visible light including PD cases. Over time symptoms can be reduced and sometimes even go away completely if implemented close enough to diagnosis or before.

A new understanding of human health from a Biophysics perspective: The human being is a hydrated carbon electrical system that has used a hydrogen burning engine powered by full spectrum sunlight throughout all of human history. The human living system stores this captured energy or charge within the water and melanin inside the body. And the mitochondria within our cells release this energy signalling and powering vital processes from clearing waste via detoxification/autophagy/apoptosis to rebuilding (growth and repair) via anabolic protein synthesis. Most diseases are caused by a lack of energy within the body rather than nuclear DNA mutations. Viewing the human body this way allows us to truly comprehend how our environment communicates health or disease to our epigenome, and how the biophysics of light operates within our human living system. It’s our environmental signals which dictate health or disease.

Our environments have changed dramatically in the past 40 years. In 2000, a study published by the U.S. Environmental Protection Agency (EPA) estimated that people in the United States spent approximately 90% of their time indoors. In 2007 the incandescent light bulb was phased out in the USA. Cold white LEDs were introduced in the early 2000’s into American homes. In 1992 the US government introduced regulation ensuring glass windows in homes block the ultraviolet, infrared and some of the red wavelengths of light from entering the home to avoid high indoor energy bills. Hundreds of countries followed suit in the subsequent years. Wi-Fi was introduced into homes in 1997 and Bluetooth in 1998. 3G was introduced in 2001, 4G in 2010 and 5G in 2019 and 6G to come in 2025. With the average American household in 2010 having 5 wirelessly connected devices and on average 25 wireless devices in 2024, the rapid saturation of pulsing frequencies in our homes, schools, workplaces and cars is becoming overwhelming to our brains and nervous systems. This is especially true for our children as each of these wireless devices have only been safety tested for ‘occasional adult use and rated to their maximum emission rating as individual devices instead of being tested as multiple wireless device exposures together. These are just some of the changes contributing to the destruction of human biological health over the past 30 years. These frequencies have never been experienced by human beings in all of human history and we explain how modern neolithic disease has exploded over recent years stemming from these dramatic alien alterations to our visible and non-visible light environment we live under. Artificial light sources in our homes pulse our eyes, skin, brains, and blood which bring on a myriad of symptoms eventually forming a disease. Disease stem from the body’s loss of energy available to repair and grow and hence diseases usually occur in the highest energy demand organs first, that being the brain and the heart. We are going to explain how the brain has changed as a result and why this alteration in bioenergetics in the human brain drives all neurodegenerative conditions.

During winter the remaining solar spectrum of UV-A, visible light (blue, green and red) and infrared are also at their weakest [12]. This reduction in energy availability forces plants and animals in these environments to hibernate, drop their photosynthetic abilities (chlorophyll in leaves for plants, and melanin in skin/coats of animals) and reduce physical exertion/activity to survive. Humans in these environments are designed to do the same whilst remaining exposed to the natural light and temperature cycles. However, we rarely follow these rules and instead become even more energy deficient during these times resulting in regular colds and build-up of toxins and unhealthy cells within the brain and body. If this goes on long enough, it can lead to a chronic disease exhibiting neurodegenerative symptoms. Further to this, if the human is exposed to - artificial light, wireless frequencies like cell phones, Wi-Fi, Bluetooth, 4G and 5G, low frequency electric and magnetic fields such as in the copper wiring of the AC power grid and solar inverters or spend 90% of their time indoors or in a car this adds another dramatic layer of stress to the biologic system and speeds up the onset of disease. These electromagnetic toxins are new to our environments and have only been widely introduced in the past 40 years. It is unequivocally what modern medicine has missed when it comes to the explosion of neurodegenerative disorders. And it is no coincidence that all of these issues such as lack of sunlight (such as vitamin d deficiency) and over exposure to artificial light and wireless frequencies (high levels of inflammation and oxidative stress) are hallmarks of neurodegeneration.

What is Parkinson’s Disease?

Parkinson's disease (PD) is a neurodegenerative disorder marked by motor and non-motor symptoms, primarily due to the loss of dopaminergic neurons in the substantia nigra, a brain area rich in iron, POMC, and neuromelanin. A PD brain is deficient in cholesterol, highly structured crystalline water networks, blood flow, omega 3’s, sunlight and vitamin d and vitamin C, which is crucial for dopamine synthesis through redox reactions. PD originates from an electrical defect in the brain linked to insufficient full-spectrum light exposure, overexposure to artificial non-native electromagnetic fields (nnEMF) such as artificial unbalanced visible light and microwave/radiofrequency or AC electric and magnetic field exposure, and a lack of seasonal nutrient dens local food and fluoride-free water. Exacerbating factors include excessive exposure to artificial light, electromagnetic fields, and wireless radiation, which degrade melanin, lead to neuron loss, and disrupt the brain’s energy balance. Over time, this affects the brain's ability to regulate physiological processes, including motor functions, leading to PD symptoms.

The disease starts far before it is diagnosed. Leading the charge of setting up the conditions for PD is being the recipient of altered biophysics signals including altered light/dark, temperature, sound, electric and magnetic fields resulting in mitochondrial dysfunction, the destruction of dopamine, melatonin, and serotonin, reduced cellular membrane charge, and increased perivascular spacing within the brain. This results in inefficient metabolism, a compromised immune system, digestive issues, REM sleep disruption, and dulled sensory perception in the thalamus. As the bioenergetics within the body’s water networks weakens detox efficiency reduces and pathogenic growth increases. An early symptom of PD is a loss of smell (anosmia) due to alpha-synuclein protein accumulation in the olfactory bulb, leading to neuronal dysfunction and loss. Motor function issues appear later, with severely impaired brain detoxification leading to chronic inflammation, oxidative stress, and pseudohypoxia.

Dopamine and melanin, both derived from the amino acid tyrosine, absorb UV light. Prolonged UV deficiency, common in those living at higher latitudes or indoors, hinders melanin production, reduces dopamine synthesis, and impairs photoelectric processes in melanin. This underscores the importance of UV light exposure for dopamine and melanin production in preventing PD. In summary, modern PD diagnosis often results from chronic sunlight deficiency, excessive artificial light at night, and excessive non-native electromagnetic fields (nnEMF). These factors compromise immune cells, allowing protein misfolding and buildup, leading to neurodegeneration.

A neurodegenerative brain has a broken energy system with respect to water, electrons, blood flow, and low voltage. The PD brain seeks repair, but environmental conditions must support this process. In PD and Alzheimer’s, low brain voltage results in low energy and intracellular dehydration, insufficient to maintain magnetic memory of the brain’s water system. The mechanism is similar across neurodegenerative diseases, but triggers determine the disease phenotype, as seen in multiple sclerosis (MS).

Mitochondrial Dysfunction in PD

Failure to maintain the healthy mitochondria is one of the early pathological events that leads to development of Parkinson’s symptoms.

Inflammation caused by the release of mitochondrial DNA may contribute to the damage found in neurodegenerative diseases such as Parkinson’s, Alzheimer’s and amyotrophic lateral sclerosis (ALS). Scientists have linked these disorders with both inflammation and an inability to properly rid cells of defective mitochondria. Mitochondria-triggered inflammation may be the missing link between the two.

Mutations in two genes associated with some forms of inherited Parkinson’s disease  PINK1 and PRKN  lead to problems in the process through which damaged mitochondria are broken down and cleared from the cell. In 2019, a group led by Richard Youle at the NINDS demonstrated that in mice with mutations in PINK1 and PRKN, inducing mitochondrial damage (either through exhaustive exercise or by altering mitochondrial DNA) activated inflammatory molecules. Those animals also lost dopamine-producing neurons in their brains and developed problems with movement hallmarks of Parkinson’s disease. These effects didn’t occur, however, when the researchers repeated the experiment with mice engineered to lack an important inflammatory molecule. Together, these findings illustrated that in animals genetically predisposed to Parkinson’s, either stress or glitches in mitochondrial DNA could trigger the inflammation that promotes the disease.

Additionally, mutations in parkin (PARK2) and PTEN-induced kinase 1 (PINK1; also known as PARK6) impede mitochondrial autophagy (mitophagy), leading to the accumulation of mitochondrial damage. This increased mtDNA somatic mutation rate degrades the mtDNA in the basal ganglion and substantia nigra, ultimately resulting in neuronal dysfunction, cell death, and movement disorders (Study24, Study26, Study27). Thus, numerous nDNA loci linked to Parkinson's disease are directly involved in modulating mitochondrial integrity and function, reinforcing the connection between mitochondrial health, inflammation, and the development of movement disorders (Study25, Study 28).

Cells have several quality control mechanisms to remove dysfunctional mitochondria. One important mechanism involves the proteins Parkin and PINK1. When a mitochondrion is damaged, PINK1 and Parkin recruit a phagophore, which engulfs the organelle and begins the process of degrading it. When such quality control systems fail, damaged mitochondrial DNA (mtDNA) can escape from the mitochondria. It appears to exit through special pores or through ruptures in the mitochondrial membrane. Once released, mtDNA fragments can activate molecules such as cGas-STING or inflammasomes, both of which sense foreign DNA from viruses and other invaders. This, in turn, can increase production of cytokines and cause inflammation.

Mitochondria have other functions that help maintain healthy brain function  or cause problems when they go awry. For example, mitochondria help control the balance of potentially toxic byproducts of cellular metabolism called reactive oxygen species and the synthesis of stress hormones like cortisol. Mitochondria are also highly dynamic  communicating with each other via signaling molecules and physical connections. They continuously undergo fission, where a large mitochondrion splits into two smaller ones, or fusion, when they combine. Taking out poorly functioning mitochondria within the brain and replacing them with well-functioning new engines is the keyway to heal and grow to a healthy vibrant expression.

Boosting mitochondrial function is essential because it ensures the presence of adequate melatonin levels, which in turn supports healthy dopamine production. Attempting to increase dopamine independently of improving mitochondrial and endogenous melatonin levels can lead to apoptotic death of striatal neurons, as unhealthy mitochondria make neurons significantly more susceptible to cytotoxicity (beware those supplementing dopamine medication). Melatonin acts as a potent free radical scavenger and is targeted to mitochondria, protecting them from endogenous and exogenous stress, including oxidative stress, and enhancing ATP production, which is positively correlated with dopamine levels. Thus, focusing on mitochondrial health not only preserves dopamine but also fundamentally transforms how melatonin functions within the body.

Cites:

• https://www.scientificamerican.com/article/could-mitochondria-be-the-key-to-a-healthy-brain/?fbclid=IwAR3lbEbnQjD_OpBHhxKlxLKDYxuKeiVijIktyaZTA-FUxxdmmBzw0ABkFRc 

• https://www.annualreviews.org/doi/10.1146/annurev-cellbio-021820-101354

Circadian Rhythm Disruptions in PD – Oxidative Stress, Pesticide Exposure, Mitochondrial Damage

• “PD patients have interrupted cycles of resting activity, changes in blood pressure, altered sleep and alertness, as well as mood and behavioral swings (Videnovic & Willis, 2016).”

• “α-synuclein, may be involved in the pathogenesis of PD (Canever et al., 2022), oxidative stress (Dias et al., 2013) and mitochondrial damage (Moon & Paek, 2015).”

• “…there is a dysregulation of the circadian cycle in PD patients that aggravates and/or stabilizes motor and non-motor symptoms during the day, depending on the stimulus received. Since circadian rhythms are controlled by a molecular clock, it is thought that these clock genes are possibly responsible for the circadian cycle dysregulation observed in PD patients.”

• “Recent studies have shown that circadian dysregulation increase the risk of developing PD, as well as, PD patients have lower expression levels of five clock genes (BMAL1, CLOCK, CRY1, PER1 and PER2) and melatonin (Leng et al., 2020; Li et al., 2021; Yang et al., 2021). It is highlighted that melatonin is a hormone in evidence used for the treatment of sleep disorders, some studies have already focused on elucidating the role of melatonin in the pathophysiology of PD (Mack et al., 2016; Pérez-Lloret & Cardinali, 2021).”

Citation

• https://onlinelibrary.wiley.com/doi/full/10.1111/jnc.15883?fbclid=IwAR1yLR54Tg0bbiWGYm33uBdcz9gPHRHhM_KEfLGualcEFgnFhRMb9ZIqVbs
  
  

The Intracellular Biologic Effect of PD – Protein Folding Issues

When a protein is constructed, there are 4 steps in protein folding. 2 instructions are controlled by the nuclear DNA and 2 are dependent upon and controlled by the mtDNA and redox state within the cell water. This includes the electric potential of the mitochondria, structural arrangement of the cytoplasmic water and charge within the cell membrane. If something causes the voltage/electric potential in the cell membrane, cytoplasm or mitochondria to be altered and/or lowered, this directly affects the energy and information available to fold a protein’s tertiary and quaternary structures. When this redox potential is lowered, it dramatically alters the atomic 3-D arrangement in the structure, and the protein does not fold properly. When this happens, it cannot self-assemble correctly to perform its physiological designed purpose.

Parkinson’s is a disease denoted by altered protein bending and ultimately folding. When proteins are shaped/constructed by non-native electromagnetic fields their 3D structure is adversely altered by this alien electrostatic charge and magnetic oscillation. If this goes on for long enough or is a dramatically acute stimulus it can result in brain damage like Parkinson’s. The brain’s safeguard against this is autophagy. Autophagy spots the proteins which have been marked by ubiquitin and recycles them, taking out the misfolded, defective ones. If autophagy is broken, this indicates dysfunction around deuterium leaking through the cell membrane into the cytoplasm and mitochondria. It also indicates free vitamin A in its aldehyde form, acting as a free radical, destroying all photoreceptors and heme proteins it can find. This occurs because non-native electromagnetic radiation breaks the covalent bond between Vitamin A and Melanopsin. A broken autophagy mechanism also indicates poor Melatonin control and hence a dysfunctional cortisol rhythm. This implies abnormal sleep and poor regeneration. It also indicates that excess artificial light and wireless radiation are environmental factors perpetuating the disease process.

All neurodegenerative diseases brains are more sensitive to the electromagnetic force. This is why nnEMF is such an environmental harm for autistic children and those with PD. All neurodegenerative diseases are also associated with low levels of ATP. ATP is an oxidiser (electron withdrawing chemical). ATP changes the charge on the side groups of proteins to control proper bending. This withdrawing event removes electrons and alters tertiary and quaternary folding of proteins. All neuro-degenerative conditions are associated with low levels of cholesterol as well.

Neurodegenerative diseases affect the brain. Cholesterol makes up a large number of membranes in the brain. Cholesterol, in sheer number, is reduced in these diseased brains, but the cholesterol that does remain is not able to have its electrons stripped to store magnetic memories. This is because its charges are altered due to changes in water chemistry around these cell membranes. Water is what allows for photon or electron flow in membranes. This means cholesterol is also a semiconductor. The polar nature of water and cholesterol molecules allows them to bond to each other in groups. Cholesterol needs electrons to remain polar. It works ideally in this state and not in its oxidized state. Brains and membranes are basically fat and water, as such, generate/store electron charge. Sulfated proteins attached to these saturated fats produce a voltage when they bind to the polar cholesterol molecule! So, when cholesterol levels in the brain are lowered, voltages drop tremendously. Cholesterol is a necessary fat that only causes trouble when it is oxidized and missing electrons, making it electron hungry!

Visible and non-visible light’s impact on the brain as it relates to Parkinson’s Disease

Since 1992 the US government changed the standards on windows blocking red and infrared light from entering indoors. Less than a decade later incandescent bulbs were banned from sale within the US rendering indoor environments almost completely red and infrared light deficient. The sun contains 51% red and infrared light with infrared being the dominant wavelength that reaches living organisms on earth. At the same time, we introduced LED lighting containing an unbalanced vapor spectrum lacking infrared and introduced on average 25 wireless devices per household. This on top of a rapid increase in power density as bandwidth (energy and information density) went from 2G in 2000 to 5G in 2020. Countries all over the world followed and now modern life is lived indoors under a complete alien spectrum of light signalling biochemical chaos within the living systems that experience it on an hourly basis. This is most detrimental with respect to health outcomes on children’s brains, pregnant mothers, fertility levels and when we sleep; leading to epidemics of infertility, childhood brain disease and insomnia.

Visible light such as artificial narrow-band blue and green light from our indoor lighting or technology screens results in the destruction of our topologic visual and non-visual photoreceptors (on our eyes and skin) making our surfaces less able to work optimally with sunlight and tell biologic time through damage to our circadian entrainment and photosynthetic systems. These visual light signals disrupt our biological clock within the hypothalamus and within every cell altering hormone release, neurotransmitter homeostasis, oxygen and nutrient delivery and waste removal, supressing melatonin, raising blood glucose independent of food and overloading the innate and adaptive immune system. Further to this, exposure to narrow-band high energy visible LED lighting (HEVL) especially in the blue light hazard between 435nm-455nm leads to mitochondrial toxic effects adversely altering the size and chape adaptability of our organelles and cells and a loss of net negative charge in our blood plasma, cerebral spinal fluid, glymphatic fluid, and alterations in the circadian sleep-wake rhythm leading to a lack of ability to regenerate and heal from the days oxidation, inflammation and hypoxia.

Non-visible light such as Non-native electromagnetic fields (nnEMF) which broadly include AC electric/magnetic fields, light flicker, dirty electricity or electromagnetic interference, microwaves, radio frequency radiation and low-level currents have a generally dehydrating effect on the body whilst stealing available energy by ensuring it remains in a sympathetic proinflammatory state. That means your Bluetooth earphones, speaker or car play system, your 5G mobile phone, your Wi-Fi router and even your robot vacuum cleaner are leading to disastrous biological effects within your human living system. This has been shown to occur via two additional mechanisms beyond the water dehydration effect within the body, they include oxidative stress cascades caused by mitochondrial free radical signalling excesses and altered calcium signalling.

nnEMF exposure results in voltage gated channel issues in the cell membranes. This leads to excessive calcium within the cell which can disrupt the synapses in the brain. Microwave Lower Freq EMFs are just one of the types of manmade EMF that is driving a brain to rapidly become neurodegenerative. As are extremely low frequency magnetic fields (ELF-MF) demonstrated by Benassi et al. who showed that ELF-MF exposure sensitizes SH-SY5Y cells to the pro-Parkinson's toxin MPP+, indicating an interaction between ELF-MF and PD neurotoxins [15]. Chemicals such as pesticides and metals like mercury and aluminium also have roles in pathogenesis of neurodegeneration, the impact of these microwave, magnetic and electric fields creating toxic vitamin A radicals in the blood destroying photoreceptors and causing an oxidative cascade is the keyway the brains immune system becomes defective early on [16].

Mitochondrial health plays a critical role in Parkinson's Disease (PD) due to its direct impact on dopamine levels. Dopamine, a crucial neurotransmitter, is both created and destroyed through various processes. As we age, particularly past 45 years old, dopamine levels drop due to the activity of the enzyme MAO-B, which exhibits fluorescence emissions when excited by blue light at 412 nm. This exposure to blue light, which destroys DHA and dopamine in the eye, frontal lobe, and brainstem, accelerates the decline in dopamine levels. This decline is exacerbated by a loss of DHA’s electrons and insufficient UV light to convert tyrosine to dopamine optimally. As a result, the loss of dopamine in the eye clock can lead to faster aging and a decrease in overall health. UV and purple light with infrared/red wavelengths can help build dopamine, while blue light destroys it more rapidly. Deprenyl and berberine can inhibit the MAO-B enzyme, promoting the development of brown fat from white adipose tissue and increasing thermogenic pathways such as UCP1, SIRT1, and AMPK. The eye governs the sympathetic nervous system (SNS), and both Deprenyl and berberine mimic the SNS effects of cold thermogenesis, raising NAD+ and increasing dopamine levels in cold environments by inactivating and quenching the MAO-B enzyme.

Photon spin numbers are (OAM) used to make matter from light. People with Parkinson’s and autistic children do best in Equatorial environments. In this environment the cells major EMF input is usually solar EMF thus control of OAM of photons within them is restored along with the damaging free radical signals are lessened and the normal free radical signals are restored. All free radicals have unpaired electrons. Understanding the quantum spin state of those free radicals is the key to the puzzle of why Parkinson’s is now explosive and why a 5G city will do the same to a non-autistic brain.

Manmade blue light and all nnEMF cause intracellular dehydration, and therefore they directly alter the bio-photon release in eukaryotic cells to cause mitochondrial diseases that change the vibrations of things inside a cell. This leads to a Warburg redox shift in many diseases. When in a Float Tank Beta-oxidation is selected for and turning off the stimulus to burn glucose as Nora Volkow and Allen Frey showed us.

Parkinson’s disease is present in a body leaking light to its environment. The amount of electromagnetic energy that you emit or absorb in your system is directly proportional to the structure of proteins in your body. Be careful with cold exposure when PD symptoms are present. Cold thermogenesis should be done at no lower temperature than 65F when submerged in water or face dunking due to a poor functioning neurological system.

RF radiation, particularly from mobile phones, has been associated with increased brain glucose metabolism in the region closest to the phone’s antenna, which could potentially lead to neuronal damage.[1] Acute RF electromagnetic radiation exposure has also been shown to impair neurogenesis and cause neuronal DNA damage in young rat brains.[2] Chronic occupational exposure to ionizing radiation, a type of RF radiation, has been associated with an increased risk of PD incidence.[3]

Wireless radiation, such as that emitted by handheld mobile and cordless phones, has been linked to an increased risk of developing glioma and acoustic neuroma, indicating potential neurotoxic effects.[4]

Artificial light, particularly over-exposure to fluorescent light, has been associated with disruption of circadian rhythms and sleep, and potential damage to dopaminergic neurons, which are primarily affected in PD. [5] Prolonged exposure to artificial light has been shown to increase neuromelanin granules in the substantia nigra and cause loss of dopaminergic neurons in animal models. Epidemiological evidence suggests a positive correlation between the prevalence of PD and light pollution.[6]

Clinical Challenges in PD

1. Low dopamine, melanin and melatonin

2. Poor mitochondrial function and dysfunctional circadian rhythm

3. Decreased blood flow to the brain

4. Poor cell membrane charge and composition (lacking cholesterol and omega 3s)

5. High homocysteine

6. Systemic inflammation

7. High mercury and led

Immune System Dysfunction in PD: What Centralized Medicine has missed 

The common misconception is that most of the current treatments to PD are not leading to good long-term outcomes. Use of L-dopa medication such as levodopa in PD beyond very short-term use poses several concerns. Neurotoxicity is one of the key issues, as preclinical studies indicate that levodopa can induce oxidative stress and mitochondrial dysfunction. Motor complications such as dyskinesia, wearing-off, and on-off phenomena are prevalent, with studies showing that 71.4% of patients on long-term therapy develop these issues, significantly impacting quality of life.

Additionally, cognitive functions can be adversely affected due to the dopamine overdose hypothesis, where exogenous dopamine can impair cognitive functions based on baseline dopamine levels in different brain regions. Furthermore, regular L-dopa administration can elevate homocysteine levels, contributing to oxidative stress and mitochondrial dysfunction, exacerbating neurodegeneration. If the body makes it, don’t take it. Simply by supplementing dopamine the body undergoes a reduced endogenous synthesis of the very little dopamine it was able to make. This can sometimes drop endogenous production almost to nothing forcing a reliance on medication and guarantee of long-term consequences related to this treatment. [study, study, study, study, study]. Many Parkinson’s patients who take an excess of L-dopa experience ‘Tachyphylaxis’ which describes an acute, sudden decrease in response to a drug after its administration, i.e. a rapid and short-term onset of drug tolerance.

When L-dopa no longer works to alleviate Parkinson’s symptoms, the next line of treatment for Parkinson’s Disease is to implement deep brain stimulation (DBS). These electrodes are implanted within the brain into the substantia nigra to electrically stimulate the brain due to the lack of sunlight through the eye and skin that the patient is lacking, and it works temporarily. But because the charge is not produced endogenously due to the poor photoelectric properties of degraded neuromelanin within this area of the brain PD symptoms usually remain. We know the therapies that help, and we know why, but the education required to elucidate the reactivation of endogenous photoelectrical communication within the human brain is not taught in medical schools leaving centralized neuro specialists with L-Dopa supplementation as an easy moneymaking temporary fix being reused for decades. The onset of PD is occurring faster than ever before in human history given the speed at which endogenous melatonin suppression, dopamine destruction and melanin degradation occurs in an environment dominated by manmade electric, magnetic and electromagnetic fields. That means it’s really easy to develop a serious brain disease in a 5G environment with 25 wireless technological devices in the average home in 2024. When a person lives around nnEMF their free electrons are being oxidized and stolen. This leaves the body’s biological surfaces (eyes, skin, gut and lungs) unable to capture sunlight as effectively.

This means oxidative environmental effects need to be removed with the most powerful in this modern world being artificial visible and non-visible light and then adding back in electrons to the biologic system via the myriads of ways of collecting electrons from the natural environment and then reintroducing powerful sunlight. Thus, reducing the destructive fields must occur first for any hope of regenerating the dopamine neurons in the substantia nigra to restore motor function and all the other symptoms of PD. This process respects that the body is in somewhat of a state of informational disorder and reorganizing needs to take place before large amounts of powerful full spectrum native energy is added and effectively accepted by the body. You can see that the solution is not just a pill or a single practice but a rational process involving some education, discipline and guidance from experts along the way if necessary.

When sunlight enters the eye, it is photoelectrically tunneled like a fibre optic transfer through to the suprachiasmatic nucleus (SCN) within the hypothalamus and onto the habenula nucleus, where the energy and information are distributed throughout the brain and body, regulating biochemistry. The tract between the retina and habenula nucleus has no synapses, indicating light conduction into the brain with photoreceptive tissue at the habenula nucleus. This tract is filled with semiconductors, water networks, photoreceptive pigments, and Mueller cells. The first synaptic stop for this photoelectrical signal after the SCN is the pituitary. When this signal is disrupted or received from an artificial unbalanced light source, this pathway and all subsequent secretions from the hypothalamus, pituitary, and prefrontal center are affected.

Individuals with Parkinson's Disease (PD) have higher incidences of melanoma and hypothyroidism due to disrupted native full spectrum light signals. When the necessary light signal is altered before reaching the hypothalamus, pituitary, or habenula nucleus, it affects the pituitary’s regulation of the thyroid, leading to hypothyroid conditions. The thyroid hormone T3 powers neuron and motor functions, explaining why PD symptoms worsen and cognitive changes occur. The degradation of melanin in the substantia nigra due to artificial EMF or lack of full spectrum sunlight results in its migration, leading to melanoma when exposed to artificial blue light and non-native EMF. The loss of neuromelanin in PD sufferers causes reduced melanin in the skin, a feature of early PD.

Solar light, rather than drugs or hormone therapies, is crucial in regulating the human adaptive immune system. In cases of Parkinson’s Disease (PD), the immune system becomes dysfunctional, leading to the accumulation of misfolded proteins in the brain. Restoring the brain’s circadian rhythm through the suprachiasmatic nucleus (SCN) within the hypothalamus can enhance mitochondrial function, reduce perivascular spaces, and improve glymphatic flow. This process helps clear alpha-synuclein, beta-amyloid, tau proteins, and other misfolded proteins, thereby restoring coherence within the brain’s water networks, including cerebrospinal fluid (CSF), blood, lymph, interstitial fluid, and cytoplasmic fluid. Such restoration improves dopamine and melatonin levels, leading to the recovery of the substantia nigra and motor cortex to a functional neuromuscular state.

To support this, it is essential to ensure that dopamine, neuromelanin, noradrenaline, melatonin, serotonin, thyroid hormone, and nicotinamide adenine dinucleotide (NAD) are restored and well regulated. This requires the eyes and skin to receive balanced ultraviolet sunlight, including a full spectrum of light rays, with 51% red and infrared light. This light excites the benzene rings within the aromatic amino acids tyrosine and tryptophan, facilitating the restoration of brain function. Additionally, consuming key nutrients found in the marine food chain, such as the omega-3 fatty acid docosahexaenoic acid (DHA) and trace minerals like magnesium, potassium, calcium, iron, zinc, copper, manganese, selenium, iodine, and sodium chloride, is crucial. These steps can revitalize the brain’s complex systems, often referred to as the "quantum computer" of the body.

Accumulating neuromelanin in the substantia nigra through disciplined exposure to full-spectrum sunlight and proper nutrition can restore dopamine-producing neurons, leading to improved executive cognition and neuromuscular function. This approach may offer significant benefits over traditional medical solutions. Before opting for drugs, supplements, diet plans, or exercise regimes, consider prioritizing light hygiene. Furthermore, while treatments like L-Dopa supplementation or deep brain stimulation (DBS)/pallidotomy may repair the substantia nigra, they do not address the skin, pituitary, SCN, or retina, meaning that PD may not be fully reversed without a comprehensive approach.

Examples of poor immune function, such as Lyme disease, Bartonella, and viral particles in the blood, indicate environmental defects rather than internal issues. Functional medicine and alternative doctors often mislead patients into unnecessary treatments, unaware of how solar light programs the adaptive immune system. Prioritize changing your light hygiene before resorting to drugs or supplements. Light controls the immune response depending on the time of day, affecting test results based on natural settings or timing. When infections arise, it suggests an environment filled with non-native electromagnetic fields (nnEMF) and blue light, causing toxic pathogens to escape and harm mitochondria.

The energy and information emitted during healing, known as biophotons, were identified by Dr. Fritz Albert Popp and later detailed by Dutch biophysicist Roeland van Wijk. These ultra-weak ultraviolet light photons, when leaked excessively, signal unwellness, revealing struggling organs through photomultiplier readings. Chronic energy and information loss lead to severe diseases, first affecting the neocortex. For adults, this very commonly manifests as Parkinson’s, Alzheimer’s, and ALS, while in children or those in utero, it appears as Autism, ADD, and ADHD. Diseases like major depressive disorder, PD, and ADHD are characterized by low dopamine levels in the brain, whereas schizophrenia involves erratic, often elevated dopamine levels, showcasing the varied expressions and behaviors stemming from dysfunctional dopamine levels.

Focusing on the PD dopamine deficit, dopamine deficit lowers the signal-to-noise ratio in a person's ability to sense their environment via the five senses integrating within the thalamus in the brain. This loss of signal-to-noise ratio prevents the body from coherently harmonizing with external environmental signals. A good example of this is our auditory sense: low dopamine makes it difficult to distinguish a person in front of you talking from the background noise, and patterns become harder to detect. With a PD dopamine deficit, following one conversation or train of thought can be challenging because all conversations or thoughts sound equally loud, making it hard to pick out a continuous stream. Instead, the unmedicated PD brain often picks up bits and pieces from several different streams of conversation or thought. Random firing neurons make up the background noise in the brain, like all the voices at a party not part of your conversation. The person whose voice you focus on, ignoring all the others, is the signal amidst the noise. When dopamine within the brain improves, the signal-to-noise ratio increases; the background noise fades, and your conversation partner’s voice becomes distinct and easy to follow. Dopamine turns up the volume on the audio signals at the party compared to the background noise, making it easier to decipher the conversation and act properly on it. This leaves people with PD open to issues with maintaining attention spans. The main factor in a low dopamine PD brain is the lack of energy available to the body to fold proteins correctly. The amount of electromagnetic energy that you emit or absorb into your system is directly proportional to the structure of proteins in your body. This is a crucial point as it demonstrates how someone with PD can continue to be unable to organize the energy and information it receives from nature into a healing, regenerative, and restorative program. This demonstrates that they may need more time in nature to reorganize their brains to begin to accept the powerful natural signals once again and optimize dopamine, melatonin, mitochondrial function, and neurological coordination once again.

Pro Tip: Vitamin C + Endogenous melatonin, dopamine and melanin synthesis via sunlight, grounding, mitigating nnEMF and HEVL + infrared light + a mitochondrial health focus = regeneration of dopaminergic neurons = optimal dopamine = PD symptom reversal.

Repairing a PD Brain

The key to repairing a PD brain involves leveraging the skin's interaction with sunlight to promote melanin creation, utilizing quantum dots (elemental minerals), and enhancing semiconductors like DHA and cholesterol. Protecting the eyes and using the skin for healing are crucial because PD patients often suffer from blue light toxicity and overexposure to artificial radiofrequency radiation. This necessitates minimizing blue light and EMF exposure using blue light blocking glasses and optimizing the home environment with the help of a building biologist.

The visual axis is weak in PD patients, making the skin a more effective medium for capturing UV light. Red and near-infrared light exposure, available during sunrise and sunset, should be used to strengthen the body's ability to harness UV light. To rebuild melanin and dopamine-producing neurons in the substantia nigra, patients should expose their skin and eyes to sunlight when the sun is above a 15-degree angle to the horizon, ensuring both UVA and UVB light exposure without causing harm. If using artificial UV lights, it is essential to pair them with red and near-infrared light to mimic natural sunlight.

Quantum dots, including essential minerals like potassium, phosphorus, and magnesium, facilitate charge movement within the body. Adequate mineral and vitamin intake, combined with consuming omega-3 fats (particularly DHA) and cholesterol, supports the brain's photoreceptive and conductive functions. These nutrients, primarily found in high-quality seasonal fats and seafood, are crucial for preconditioning the body for full-spectrum sunlight.

The skin and brain originate from the neuroectoderm in an embryo, linking them intrinsically and suggesting a pathway for brain repair via the skin. Stimulating melanin creation with UVA light and ensuring a healthy redox state by building Vitamin D through UVB exposure are vital. Melanin captures UV photons, and its interaction with DHA facilitates efficient electron transport within the body, making melanin an ideal semiconductor.

In summary, repairing a PD brain involves controlling light exposure to the eyes and skin, ensuring adequate intake of essential minerals and fats, and leveraging the skin's natural photoreceptive properties to enhance brain function and mitigate PD symptoms.

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